Citation: | Chen Zhongbao, Zhou Jiangqiao, Qiu Tao, et al. Effect of pretreatment with allopurinol on the expression of Bax, Bcl-2 and Caspase-3 in kidney of ischemia-reperfusion injury rats[J]. ORGAN TRANSPLANTATION, 2016, 7(2): 139-143. doi: 10.3969/j.issn.1674-7445.2016.02.012 |
[1] |
乔良伟, 曲青山.传统尸体供肾与心脏死亡器官捐献供肾肾移植效果的比较[J].器官移植, 2013, 4(5): 284-287. http://www.organtranspl.com/browse/detail/qkid/66/id/271.html
Qiao LW, Qu QS. Comparison of the effect of renal transplantation between traditional cadaver donor and donation after cardiac death[J]. Organ Transplant, 2013, 4(5):284-287. http://www.organtranspl.com/browse/detail/qkid/66/id/271.html
|
[2] |
李小庆, 程颖, 刘强, 等.心脏死亡器官捐献供肾移植早期肾功能恢复情况的荟萃分析[J].中华器官移植杂志, 2013, 34(3):167-170. http://med.wanfangdata.com.cn/Paper/Detail/PeriodicalPaper_zhqgyz98201303011
Li XQ, Cheng Y, Liu Q, et al. Meta-analysis of the early renal function recovery after kidney transplant from cardiac death donors[J]. Chin J Organ Transplant, 2013, 34(3):167-170. http://med.wanfangdata.com.cn/Paper/Detail/PeriodicalPaper_zhqgyz98201303011
|
[3] |
Lee WY, Koh EJ, Lee SM. A combination of ischemic preconditioning and allopurinol protects against ischemic injury through a nitric oxide-dependent mechanism[J]. Nitric Oxide, 2012, 26(1):1-8. doi: 10.1016/j.niox.2011.11.002
|
[4] |
Neuhof C, Neuhof H. Calpain system and its involvement in myocardial ischemia and reperfusion injury[J]. World J Cardiol, 2014, 6(7):638-652. doi: 10.4330/wjc.v6.i7.638
|
[5] |
Miyazawa K, Miyagi S, Maida K, et al. Edaravone, a free radical scavenger, improves the graft viability on liver transplantation from non-heart-beating donors in pigs[J]. Transplant Proc, 2014, 46(4):1090-1094. doi: 10.1016/j.transproceed.2013.11.155
|
[6] |
Jennings RB. Historical perspective on the pathology of myocardial ischemia/reperfusion injury[J]. Circ Res, 2013, 113(4):428-438. doi: 10.1161/CIRCRESAHA.113.300987
|
[7] |
Kaul S. The "no reflow" phenomenon following acute myocardial infarction: mechanisms and treatment options[J]. J Cardiol, 2014, 64(2):77-85. doi: 10.1016/j.jjcc.2014.03.008
|
[8] |
Gutowski M, Kowalczyk S. A study of free radical chemistry: their role and pathophysiological significance[J]. Acta Biochim Pol, 2013, 60(1):1-16. http://www.ncbi.nlm.nih.gov/pubmed/23513192
|
[9] |
Gormus ZI, Celik JB, Ergene N, et al. Does preoperative administration of allopurinol protect the lungs from ischemia-reperfusion injury occuring during cardiopulmonary bypass[J]. Bratisl Lek Listy, 2013, 114(10):561-565. http://www.ncbi.nlm.nih.gov/pubmed/24156678
|
[10] |
Rachmat FD, Rachmat J, Sastroasmoro S, et al. Effect of allopurinol on oxidative stress and hypoxic adaptation response during surgical correction of tetralogy of fallot[J]. Acta Med Indones, 2013, 45(2):94-100. https://www.researchgate.net/publication/239732567_Effect_of_allopurinol_on_oxidative_stress_and_hypoxic_adaptation_response_during_surgical_correction_of_tetralogy_of_fallot
|
[11] |
Sapalidis K, Papavramidis TS, Gialamas E, et al. The role of allopurinol's timing in the ischemia reperfusion injury of small intestine[J]. J Emerg Trauma Shock, 2013, 6(3):203-208. doi: 10.4103/0974-2700.115346
|
[12] |
Renault TT, Teijido O, Antonsson B, et al. Regulation of Bax mitochondrial localization by Bcl-2 and Bcl-x(L): keep your friends close but your enemies closer[J]. Int J Biochem Cell Biol, 2013, 45(1):64-67. doi: 10.1016/j.biocel.2012.09.022
|
[13] |
Yang T, Zhang Y, Li Y, et al. High amounts of fluoride induce apoptosis/cell death in matured ameloblast-like LS8 cells by downregulating Bcl-2[J]. Arch Oral Biol, 2013, 58(9):1165-1173. doi: 10.1016/j.archoralbio.2013.03.016
|
[14] |
Renault TT, Manon S. Bax: addressed to kill[J]. Biochimie, 2011, 93(9):1379-1391. doi: 10.1016/j.biochi.2011.05.013
|
[15] |
Taeger CD, Müller-Seubert W, Horch RE, et al. Ischaemia-related cell damage in extracorporeal preserved tissue: new findings with a novel perfusion model[J]. J Cell Mol Med, 2014, 18(5):885-894. doi: 10.1111/jcmm.2014.18.issue-5
|
[16] |
Snigdha S, Smith ED, Prieto GA, et al. Caspase-3 activation as a bifurcation point between plasticity and cell death[J]. Neurosci Bull, 2012, 28(1):14-24. doi: 10.1007/s12264-012-1057-5
|